DEMBARAH 200

Dengue is an arbovirus disease caused by four viruses that are related to the same viruses which cause Yellow Fever and St. Louis encephalitis. Dengue viruses are transmitted from person to person by the Aedes Aegypti and Albopictus (tiger) mosquitoes.

Periodic epidemics have occurred in the Western Hemisphere for over 200 years but the last 15 years has seen an alarming increase in large epidemics in many tropical countries. Since the 1990s there have been large-scale epidemics in Singapore, Indonesia, Thailand, Philippines, India, Vietnam and Brazil, which have caused many thousands of deaths.

The first time an individual is infected by a mosquito then classical or primary dengue will ensue: if this person is bitten again, even months later, the resulting disease is known as a secondary dengue infection which may develop into Dengue Haemorrhagic Fever (DHF).

Primary dengue (Classical Dengue) is characterised by acute high-grade fever, frontal headache, retro-orbital pain, myalgias, arthralgias (acute muscular and joint pain) and often a maculopapular rash. Many patients also notice a change in taste sensation. Clinically, the illness may be indistinguishable from influenza, measles, or rubella. The acute phase may last for up to one week with a prolonged convalescence characterised by weakness, anaemia, anorexia and general malaise.

Secondary dengue infection or Dengue Haemorrhagic Fever (DHF) may resemble classical dengue or other viral syndromes in the first few days of the illness. DHF may cause fever lasting 2 to 7 days and a variety of nonspecific symptoms. As the fever begins to break the patient becomes restless, lethargic and may show signs of circulatory failure and develop haemorrhagic symptoms.

Most patients develop thrombocytopenia – a significant reduction in the number of thrombocytes or platelets. These cells cause the blood to coagulate. The condition results in skin haemorrhages, bleeding gums, nosebleeds, vomiting and diahorrea. Internal bleeding from the liver, bowel and stomach may also occur. Patients with these symptoms are known to have Dengue Shock Syndrome (DSS). Early signs of DSS are restlessness; cold clammy skin; a rapid weak pulse; a reduction in blood pressure and/or hypertension.

Dengue Hemorrhagic Fever/DSS is most severe in children under 15 years, but it can also seriously affect adults. Mortality rates can be kept below 1% with early diagnosis and fluid replacement therapy, however, the key to survival is early diagnosis. Patients with suspected dengue fever should be given acetaminophen preparations for fever, and not aspirin due to its anticoagulant properties.

Dembarah200 is an alternative treatment that comes from the homoeopathic system of medicine. This system was perfected in Germany in 1796. In this system of medicine the human immune system is stimulated and strengthened to respond to specific diseases and medical problems.

The way a homoeopathic medicine functions is very similar to a vaccine, however the effect on the immune system is much deeper. Achieving this effect on the human immune system requires very careful selection of specific plants and other natural components.

Once selected, these components are processed using the unique homoeopathic process that extracts and amplifies the deep healing powers stored within the molecules of each herbal component. Chemical anti-viral agents do not assist the natural healing processes of the human body; instead they act chemically on the virus as a controlled poison.

Chemical based drugs are designed to poison viruses and bacteria in the hope that they will be killed before the drugs begin to cause harmful side effects to the body’s organs. However, in the case of dengue no effective conventional drugs or vaccines currently exist to treat this virus.

Pharmacology: Dembarah200 works as a powerful allergen at the molecular level to cause a rapid mobilization of the immune system against the dengue virus. The dengue virus is a formidable organism because of its ability to confuse the human immune system. If a person has had dengue, specific antibodies attach to the outer envelope of the microbe virus. In dengue secondary infection these antibodies are confused by the newly retrofitted dengue virus.

The confused antibodies pass incorrect signals to virus hunting macrophages of the immune system. When the macrophages arrive to eat the virus, dengue is consumed but does not die. Dengue then takes control, and through the immune system, enters every organ in the human body. The confused immune system tries to overcome the infection by inducing fever. Fluids and electrolytes are thus lost.

Haemorrhaging occurs when the virus decreases coagulation control by killing off most of the thrombocytes, also known as platelets. Death then occurs from Dengue Shock Syndrome. The components of Dembarah200 work together in a synergistic way as a powerful allergen to stimulate an optimum response from the human immune system to the dengue threat. The liver is stimulated to expel the virus into the bowel. Fever control is also achieved through action on the hypothalamus.

Simultaneously the thymus gland and bone marrow are also stimulated to produce much larger numbers of antibodies and virus-hunting macrophages than would normally occur. The result is that the virus is overwhelmed by the immune system response An exact description of the action of each component is difficult to determine since the formula achieves its unique effect only through synergistic interaction between all of the components.